2a58488c0b387e68df38470e8f248b0d8cc13e28
ARID1A.md
| ... | ... | @@ -3,7 +3,7 @@ bibliography: 'morinlab.bib' |
| 3 | 3 | csl: 'NLM.csl' |
| 4 | 4 | link-citations: true |
| 5 | 5 | nocite: | |
| 6 | - @wienandGenomicAnalysesFlowsorted2019, @rossiCodingGenomeSplenic2012, @loveGeneticLandscapeMutations2012, @zhangGeneticHeterogeneityDiffuse2013, @reddyGeneticFunctionalDrivers2017, @hubschmannMutationalMechanismsShaping2021, @krysiakRecurrentSomaticMutations2017 |
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| 6 | + @barisicARID1AOrchestratesSWI2024, @wienandGenomicAnalysesFlowsorted2019, @rossiCodingGenomeSplenic2012, @loveGeneticLandscapeMutations2012, @zhangGeneticHeterogeneityDiffuse2013, @reddyGeneticFunctionalDrivers2017, @hubschmannMutationalMechanismsShaping2021, @krysiakRecurrentSomaticMutations2017 |
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| 9 | 9 | [[_TOC_]] |
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| 13 | 13 | ARID1A (AT-rich interactive domain-containing protein 1A) is a gene that encodes a subunit of the SWI/SNF chromatin remodeling complex, which is involved in regulating DNA accessibility. Mutations in ARID1A are implicated in various cancers, including B-cell lymphomas. They are the most abundant in Burkitt lymphoma but also occur in FL and, to a lesser extent, DLBCL. |
| 14 | 14 | Overall, components of SWI/SNF have been identified as an emerging theme in germinal centre-derived B-cell lymphomas but their role has not been thoroughly elucidated.[@lunningMutationChromatinModifiers2015] |
| 15 | 15 | |
| 16 | +## Experimental Evidence |
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| 17 | + |
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| 18 | +Driver mutations in this gene typically resutl in a loss of function (LOF).[@barisicARID1AOrchestratesSWI2024] |
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| 16 | 19 | |
| 17 | 20 | ## Relevance tier by entity |
| 18 | 21 |