4efcd2574d847e0d778264dd5861c863f76709d2
CD79B.md
| ... | ... | @@ -20,7 +20,7 @@ This and other common mutations primarily occur in the immunoreceptor tyrosine-b |
| 20 | 20 | |
| 21 | 21 | |Entity|Tier|Description | |
| 22 | 22 | |:------:|:----:|--------------------------------------| |
| 23 | -| |1 |high-confidence DLBCL gene with functional evidence[@davisChronicActiveBcellreceptor2010] [@morinFrequentMutationHistonemodifying2011]| |
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| 23 | +| |1 |high-confidence DLBCL gene with functional evidence[@davisChronicActiveBcellreceptor2010;@morinFrequentMutationHistonemodifying2011]| |
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| 24 | 24 | | |2 |relevance in FL not firmly established| |
| 25 | 25 | | |3 |Retired, Failed QC[@paneaWholeGenomeLandscape2019]| |
| 26 | 26 | |
| ... | ... | @@ -35,7 +35,7 @@ This and other common mutations primarily occur in the immunoreceptor tyrosine-b |
| 35 | 35 | |
| 36 | 36 | ## CD79B Hotspots |
| 37 | 37 | |
| 38 | -Mutations at Y196 enhance B-cell receptor (BCR) signaling by preventing the negative regulatory feedback provided by Lyn kinase, a feedback inhibitor of BCR signaling. This results in continuous activation of the NF-κB pathway, promoting tumor cell survival and proliferation.<sup>4</sup> |
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| 38 | +Mutations at Y196 enhance B-cell receptor (BCR) signaling by preventing the negative regulatory feedback provided by Lyn kinase, a feedback inhibitor of BCR signaling. This results in continuous activation of the NF-κB pathway, promoting tumor cell survival and proliferation.<sup>[@kimCD79BMYD88Mutations2014]</sup> |
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| 39 | 39 | |
| 40 | 40 | | Chromosome |Coordinate (hg19) | ref>alt | HGVSp | |
| 41 | 41 | | :---:| :---: | :--: | :---: | |