Changes in 5934507: update

				morinlab.bib
			
          @@ -1,3 +1,22 @@

          +@article{qiuIRF8mutantCellLymphoma2024,

          +  title = {{{IRF8-mutant B}} Cell Lymphoma Evades Immunity through a {{CD74-dependent}} Deregulation of Antigen Processing and Presentation in {{MHCII}} Complexes},

          +  author = {Qiu, Zhijun and Khalife, Jihane and Ethiraj, Purushoth and Jaafar, Carine and Lin, An-Ping and Holder, Kenneth N. and Ritter, Jacob P. and Chiou, Lilly and Huelgas-Morales, Gabriela and Aslam, Sadia and Zhang, Zhao and Liu, Zhijie and Arya, Shailee and Gupta, Yogesh K. and Dahia, Patricia L. M. and Aguiar, Ricardo C. T.},

          +  date = {2024-07-12},

          +  journaltitle = {Science Advances},

          +  shortjournal = {Sci Adv},

          +  volume = {10},

          +  number = {28},

          +  eprint = {38996030},

          +  eprinttype = {pmid},

          +  pages = {eadk2091},

          +  issn = {2375-2548},

          +  doi = {10.1126/sciadv.adk2091},

          +  abstract = {The mechanism by which interferon regulatory factor 8 (IRF8) mutation contributes to lymphomagenesis is unknown. We modeled IRF8 variants in B cell lymphomas and found that they affected the expression of regulators of antigen presentation. Expression of IRF8 mutants in murine B cell lymphomas suppressed CD4, but not CD8, activation elicited by antigen presentation and downmodulated CD74 and human leukocyte antigen (HLA) DM, intracellular regulators of antigen peptide processing/loading in the major histocompatibility complex (MHC) II. Concordantly, mutant IRF8 bound less efficiently to the promoters of these genes. Mice harboring IRF8 mutant lymphomas displayed higher tumor burden and remodeling of the tumor microenvironment, typified by depletion of CD4, CD8, and natural killer cells, increase in regulatory T cells and T follicular helper cells. Deconvolution of bulk RNA sequencing data from IRF8-mutant human diffuse large B cell lymphoma (DLBCL) recapitulated part of the immune remodeling detected in mice. We concluded that IRF8 mutations contribute to DLBCL biology by facilitating immune escape.},

          +  langid = {english},

          +  pmcid = {PMC11244530},

          +  keywords = {Animals,Antigen Presentation,Antigens Differentiation B-Lymphocyte,Cell Line Tumor,Gene Expression Regulation Neoplastic,Histocompatibility Antigens Class II,Humans,Interferon Regulatory Factors,Lymphoma B-Cell,Lymphoma Large B-Cell Diffuse,Mice,Mutation,Tumor Escape,Tumor Microenvironment}

          +}

          +

           @article{guoSGK1MutationStatus2022b,

             title = {{{SGK1}} Mutation Status Can Further Stratify Patients with Germinal Center {{B-cell-like}} Diffuse Large {{B-cell}} Lymphoma into Different Prognostic Subgroups},

             author = {Guo, Baoping and Huang, Yujie and Duan, Ying and Liao, Chengcheng and Cen, Hong},

...	...	@@ -1,3 +1,22 @@
	1	+@article{qiuIRF8mutantCellLymphoma2024,
	2	+ title = {{{IRF8-mutant B}} Cell Lymphoma Evades Immunity through a {{CD74-dependent}} Deregulation of Antigen Processing and Presentation in {{MHCII}} Complexes},
	3	+ author = {Qiu, Zhijun and Khalife, Jihane and Ethiraj, Purushoth and Jaafar, Carine and Lin, An-Ping and Holder, Kenneth N. and Ritter, Jacob P. and Chiou, Lilly and Huelgas-Morales, Gabriela and Aslam, Sadia and Zhang, Zhao and Liu, Zhijie and Arya, Shailee and Gupta, Yogesh K. and Dahia, Patricia L. M. and Aguiar, Ricardo C. T.},
	4	+ date = {2024-07-12},
	5	+ journaltitle = {Science Advances},
	6	+ shortjournal = {Sci Adv},
	7	+ volume = {10},
	8	+ number = {28},
	9	+ eprint = {38996030},
	10	+ eprinttype = {pmid},
	11	+ pages = {eadk2091},
	12	+ issn = {2375-2548},
	13	+ doi = {10.1126/sciadv.adk2091},
	14	+ abstract = {The mechanism by which interferon regulatory factor 8 (IRF8) mutation contributes to lymphomagenesis is unknown. We modeled IRF8 variants in B cell lymphomas and found that they affected the expression of regulators of antigen presentation. Expression of IRF8 mutants in murine B cell lymphomas suppressed CD4, but not CD8, activation elicited by antigen presentation and downmodulated CD74 and human leukocyte antigen (HLA) DM, intracellular regulators of antigen peptide processing/loading in the major histocompatibility complex (MHC) II. Concordantly, mutant IRF8 bound less efficiently to the promoters of these genes. Mice harboring IRF8 mutant lymphomas displayed higher tumor burden and remodeling of the tumor microenvironment, typified by depletion of CD4, CD8, and natural killer cells, increase in regulatory T cells and T follicular helper cells. Deconvolution of bulk RNA sequencing data from IRF8-mutant human diffuse large B cell lymphoma (DLBCL) recapitulated part of the immune remodeling detected in mice. We concluded that IRF8 mutations contribute to DLBCL biology by facilitating immune escape.},
	15	+ langid = {english},
	16	+ pmcid = {PMC11244530},
	17	+ keywords = {Animals,Antigen Presentation,Antigens Differentiation B-Lymphocyte,Cell Line Tumor,Gene Expression Regulation Neoplastic,Histocompatibility Antigens Class II,Humans,Interferon Regulatory Factors,Lymphoma B-Cell,Lymphoma Large B-Cell Diffuse,Mice,Mutation,Tumor Escape,Tumor Microenvironment}
	18	+}
	19	+
1	20	@article{guoSGK1MutationStatus2022b,
2	21	title = {{{SGK1}} Mutation Status Can Further Stratify Patients with Germinal Center {{B-cell-like}} Diffuse Large {{B-cell}} Lymphoma into Different Prognostic Subgroups},
3	22	author = {Guo, Baoping and Huang, Yujie and Duan, Ying and Liao, Chengcheng and Cen, Hong},