delete extraneous refs

-@article{liFastAccurateLongread2010,

- title = {Fast and Accurate Long-Read Alignment with {{Burrows}}–{{Wheeler}} Transform},

- author = {Li, Heng and Durbin, Richard},

- date = {2010-03-01},

- journaltitle = {Bioinformatics},

- shortjournal = {Bioinformatics},

- volume = {26},

- number = {5},

- pages = {589--595},

- issn = {1367-4803},

- doi = {10.1093/bioinformatics/btp698},

- url = {https://academic.oup.com/bioinformatics/article/26/5/589/211735},

- urldate = {2019-07-02},

- abstract = {Abstract. Motivation: Many programs for aligning short sequencing reads to a reference genome have been developed in the last 2 years. Most of them are very ef},

- langid = {english}

-}

-

-@article{liHNRNPH1RequiredRhabdomyosarcoma2018,

- title = {{{HNRNPH1}} Is Required for Rhabdomyosarcoma Cell Growth and Survival},

- author = {Li, Yanfeng and Bakke, Jesse and Finkelstein, David and Zeng, Hu and Wu, Jing and Chen, Taosheng},

- date = {2018-01-24},

- journaltitle = {Oncogenesis},

- volume = {7},

- number = {1},

- pages = {1--13},

- publisher = {Nature Publishing Group},

- issn = {2157-9024},

- doi = {10.1038/s41389-017-0024-4},

- url = {https://www.nature.com/articles/s41389-017-0024-4},

- urldate = {2022-09-28},

- abstract = {Rhabdomyosarcoma (RMS) is an aggressive and difficult to treat cancer characterized by a muscle-like phenotype. Although the average 5-y survival rate is 65\% for newly diagnosed RMS, the treatment options for metastatic disease are limited in efficacy, with the 5-y survival rate plummeting to 30\%. Heterogenous nuclear ribonucleoprotein H1 (HNRNPH1) is an RNA-binding protein that is highly expressed in many cancers, including RMS. To determine the role HNRNPH1 plays in RMS tumorigenesis, we investigated its expression and effect on growth in three cellular models of RMS: RD, RH30, and RH41 cells. Upon knockdown of HNRNPH1, growth of all cell lines was reduced, most likely through a combination of apoptosis and cell cycle arrest. We then recapitulated this finding by performing in vivo xenograft studies, in which knockdown of HNRNPH1 resulted in a reduction of tumor formation and growth. We used RNA sequencing to identify changes in gene expression after HNRNPH1 knockdown and found altered splicing of some oncogenes. Our data contribute to understanding the role of HNRNPH1 in RMS development.},

- issue = {1},

- langid = {english},

- keywords = {Cell growth,Sarcoma}

-}

-

-@article{limEffectModulationHnRNP2010,

- title = {Effect of {{Modulation}} of {{hnRNP L Levels}} on the {{Decay}} of Bcl-2 {{mRNA}} in {{MCF-7 Cells}}},

- author = {Lim, Mi-Hyun and Lee, Dong-Hyoung and Jung, Seung Eun and Youn, Dong-Ye and Park, Chan Sun and Lee, Jeong-Hwa},

- date = {2010-02},

- journaltitle = {The Korean Journal of Physiology \& Pharmacology: Official Journal of the Korean Physiological Society and the Korean Society of Pharmacology},

- shortjournal = {Korean J Physiol Pharmacol},

- volume = {14},

- number = {1},

- eprint = {20221275},

- eprinttype = {pmid},

- pages = {15--20},

- issn = {2093-3827},

- doi = {10.4196/kjpp.2010.14.1.15},

- abstract = {It has been shown that CA repeats in the 3'-untranslated region (UTR) of bcl-2 mRNA contribute the constitutive decay of bcl-2 mRNA and that hnRNP L (heterogenous nuclear ribonucleoprotein L) interacts with CA repeats in the 3'-UTR of bcl-2 mRNA, both in vitro and in vivo. The aim of this study was to determine whether the alteration of hnRNP L affects the stability of bcl-2 mRNA in vivo. Human breast carcinoma MCF-7 cells were transfected with hnRNP L-specific shRNA or hnRNP L-expressing vector to decrease or increase hnRNP L levels, respectively, followed by an actinomycin D chase. An RT-PCR analysis showed that the rate of degradation of endogenous bcl-2 mRNA was not affected by the decrease or increase in the hnRNP L levels. Furthermore, during apoptosis or autophagy, in which bcl-2 expression has been reported to decrease, no difference in the degradation of bcl-2 mRNA was observed between control and hnRNP L-knock down MCF-7 Cells. On the other hand, the levels of AUF-1 and nucleolin, transacting factors for ARE in the 3'UTR of bcl-2 mRNA, were not significantly affected by the decrease in hnRNP L, suggesting that a disturbance in the quantitative balance between these transacting factors is not likely to interfere with the effect of hnRNP L. Collectively, the findings indicate that the decay of bcl-2 mRNA does not appear to be directly controlled by hnRNP L in vivo.},

- langid = {english},

- pmcid = {PMC2835978},

- keywords = {bcl-2 mRNA stability,hnRNP L}

-}

-

-@article{limFcGammaReceptor2011,

- title = {Fc Gamma Receptor {{IIb}} on Target {{B}} Cells Promotes Rituximab Internalization and Reduces Clinical Efficacy},

- author = {Lim, S H and Vaughan, A T and Ashton-Key, M and Williams, E L and Dixon, S V and Chan, H T C and Beers, S A and French, R R and Cox, K L and Davies, A J and Potter, K N and Mockridge, C I and Oscier, D G and Johnson, P W M and Cragg, M S and Glennie, M J},

- date = {2011-08},

- journaltitle = {Blood},

- volume = {118},

- number = {9},

- pages = {1--12}

-}

-@article{limMantleCellLymphoma2010,

- title = {The {{Mantle Cell Lymphoma International Prognostic Index}}: {{Does}} It Work in Routine Practice?},

- shorttitle = {The {{Mantle Cell Lymphoma International Prognostic Index}}},

- author = {Lim, S. Y. and Horsman, J. M. and Hancock, B. W.},

- date = {2010-01},

- journaltitle = {Oncology Letters},

- shortjournal = {Oncol Lett},

- volume = {1},

- number = {1},

- eprint = {22966280},

- eprinttype = {pmid},

- pages = {187--188},

- issn = {1792-1074},

- doi = {10.3892/ol_00000034},

- abstract = {The Mantle Cell Lymphoma International Prognostic Index (MIPI) combines four factors to differentiate low-, intermediate- and high-risk prognostic groups in advanced mantle cell lymphoma using data from patients treated in clinical trials. To evaluate its use in routine practice, we applied the simplified index retrospectively to 50 consecutive new patients attending our lymphoma service. In the log-rank and multiple comparison statistical tests there was favorable differentiation between survival curves, and particularly between the high- and low-risk groups. We concluded that the MIPI is of value in routine lymphoma practice.},

- langid = {english},

- pmcid = {PMC3436357}

-}

-

-@article{limMisalignedSequencingReads2022,

- title = {Misaligned Sequencing Reads from the {{GNAQ-pseudogene}} Locus May Yield {{GNAQ}} Artefact Variants},

- author = {Lim, Jing Quan and Lim, Soon Thye and Ong, Choon Kiat},

- date = {2022-01-24},

- journaltitle = {Nature Communications},

- shortjournal = {Nat Commun},

- volume = {13},

- number = {1},

- pages = {458},

- publisher = {Nature Publishing Group},

- issn = {2041-1723},

- doi = {10.1038/s41467-022-28115-z},

- url = {https://www.nature.com/articles/s41467-022-28115-z},

- urldate = {2022-05-20},

- issue = {1},

- langid = {english},

- keywords = {Cancer,Computational biology and bioinformatics,Genetics}

-}

-

-@article{liNanoparticleconjugatedAptamerTargeting2015,

- title = {Nanoparticle-Conjugated Aptamer Targeting {{hnRNP A2}}/{{B1}} Can Recognize Multiple Tumor Cells and Inhibit Their Proliferation},

- author = {Li, Hui and Guo, Lei and Huang, Aixue and Xu, Hua and Liu, Xuemei and Ding, Hongmei and Dong, Jie and Li, Jie and Wang, Chaonan and Su, Xueting and Ge, Xingfeng and Sun, Leqiao and Bai, Chenjun and Shen, Xuelian and Fang, Tao and Li, Zhanghua and Zhou, Yong and Zhan, Linsheng and Li, Shaohua and Xie, Jianwei and Shao, Ningsheng},

- date = {2015-09-01},

- journaltitle = {Biomaterials},

- shortjournal = {Biomaterials},

- volume = {63},

- pages = {168--176},

- issn = {0142-9612},

- doi = {10.1016/j.biomaterials.2015.06.013},

- url = {https://www.sciencedirect.com/science/article/pii/S014296121500527X},

- urldate = {2022-10-14},

- abstract = {In this study, we further investigated a previously developed aptamer targeting ROS 17/2.8 (rat osteosarcoma) cells. We found that this C6-8 aptamer specifically binds to heterogeneous nuclear ribonucleoprotein (hnRNP) A2/B1 and that it specifically labeled multiple tumor-cell lines as effectively as hnRNP A2/B1 monoclonal antibodies. When conjugated with fluorescent carbon nanodots (CDots) it could freely enter multiple living tumor cell lines (HepG2, MCF-7, H1299, and HeLa), whose growth it inhibited by targeting hnRNP A2/B1. Similar inhibitory effects were observed when the GFP-HepG2 hepatocarcinoma cells treated with C6-8-conjugated CDots were implanted in nude mice. Our work provides a new aptamer for targeting/labeling multiple tumor cell types, and its nanoparticle conjugates bring further advantages that increase its potential for use in cancer diagnosis and therapy.},

- langid = {english},

- keywords = {Aptamer,Heterogeneous nuclear ribonucleoprotein A2/B1,Nanoparticles,SELEX,Tumor cells}

-}

-@article{linGenomeDynamicsHuman2014,

- title = {Genome Dynamics of the Human Embryonic Kidney 293 Lineage in Response to Cell Biology Manipulations},

- author = {Lin, Yao-Cheng and Boone, Morgane and Meuris, Leander and Lemmens, Irma and Roy, Nadine Van and Soete, Arne and Reumers, Joke and Moisse, Matthieu and Plaisance, Stéphane and Drmanac, Radoje and Chen, Jason and Speleman, Frank and Lambrechts, Diether and family=Peer, given=Yves Van, prefix=de, useprefix=false and Tavernier, Jan and Callewaert, Nico},

- date = {2014-09-03},

- journaltitle = {Nature Communications},

- shortjournal = {Nat Commun},

- volume = {5},

- number = {1},

- pages = {1--12},

- issn = {2041-1723},

- doi = {10.1038/ncomms5767},

- url = {https://www.nature.com/articles/ncomms5767},

- urldate = {2019-12-24},

- abstract = {The human embryonic kidney 293 (HEK293) cell lineage is widely used in cell biology and biotechnology. Here, the authors apply whole genome resequencing methods to characterise genomic variation in six HEK293 cell lines and suggest that this variation could affect experiments using these cell lines.},

- langid = {english}

-}

-